Pharmacological Reference · v2

Anabolic–Androgenic Steroid Knowledge Graph

Click any compound → profile loads in panel · Use ranking buttons to sort by hair / gyno / cardiac / organ risk
Testosterone
DHT-Derived
19-Nor Class
High Hepatotoxicity
Aromatizes
Non-Aromatizing
Level 1 — Base Hormone
Testosterone · Base Hormone
Testosterone
Endogenous androgen · Reference standard
AromatizesInjectable
Anabolic
100
Androgenic
100
BulkingStrengthRecompTRT
✂ MODERATE⚠ MODERATE
TESTOSTERONE ESTERS
Level 2A — Testosterone Esters
Testosterone Ester
Testosterone Cypionate
Test C · Depo-Testosterone
AromatizesInjectable
Anabolic
100
Androgenic
100
BulkingStrengthTRT
✂ MODERATE⚠ MODERATE
Testosterone Ester
Testosterone Enanthate
Test E · Delatestryl
AromatizesInjectable
Anabolic
100
Androgenic
100
BulkingStrengthTRT
✂ MODERATE⚠ MODERATE
Testosterone Ester
Testosterone Propionate
Test P · Testoviron
AromatizesInjectable
Anabolic
100
Androgenic
100
CuttingRecompTRT
✂ MODERATE⚠ LOW
DHT DERIVATIVES
Level 2B — DHT-Derived Compounds
DHT-Derived
Stanozolol
Winstrol · Winny
Non-AromatizingHepatotoxic
Anabolic
320
Androgenic
30
CuttingStrength
✂ VERY HIGH⚠ NONE
DHT-Derived
Masteron
Drostanolone P/E
Non-AromatizingInjectable
Anabolic
62
Androgenic
25
CuttingRecomp
✂ VERY HIGH⚠ NONE
DHT-Derived · Oral
Oxandrolone
Anavar · Var
Non-AromatizingHepatotoxic
Anabolic
322–630
Androgenic
24
CuttingStrengthRecomp
✂ HIGH⚠ NONE
DHT-Derived · Oral
Proviron
Mesterolone
Non-AromatizingOral
Anabolic
40
Androgenic
30–40
CuttingRecomp
✂ HIGH⚠ NONE
DHT-Derived · ⚠ Very High Risk
Halotestin
Fluoxymesterone
Non-AromatizingSevere Hepatotoxicity⚠ Extreme
Anabolic
1900
Androgenic
850
Strength
✂ EXTREME⚠ NONE
19-NOR DERIVATIVES
Level 2C — 19-Nor (Nandrolone) Class
19-Nor Derivative
Nandrolone Decanoate
Deca-Durabolin · Deca
Aromatizes (low)Injectable
Anabolic
125
Androgenic
37
BulkingStrengthRecomp
✂ LOW⚠ HIGH
19-Nor Derivative
Nandrolone Phenylprop.
NPP · Durabolin
Aromatizes (low)Injectable
Anabolic
125
Androgenic
37
BulkingRecomp
✂ LOW⚠ HIGH
19-Nor · ⚠ High Risk
Trenbolone
Tren A / Tren E / Parabolan
Non-Aromatizing⚠ High Risk
Anabolic
500
Androgenic
500
BulkingCuttingStrength
✂ VERY HIGH⚠ VERY HIGH
ORAL AAS
Level 2D — Oral AAS (17α-alkylated)
Oral AAS · 17α-AA
Methandrostenolone
Dianabol · Dbol
AromatizesHepatotoxic
Anabolic
210
Androgenic
60
BulkingStrength
✂ MODERATE⚠ HIGH
Oral AAS · 17α-AA · ⚠ Very High Risk
Oxymetholone
Anadrol · A-Bombs
Estrogenic (indirect)Severe Hepatotoxicity
Anabolic
320
Androgenic
45
BulkingStrength
✂ MODERATE⚠ HIGH
Base Hormone
Testosterone
Endogenous androgen · Reference molecule
OVERALL RISK: Moderate
Overview
Hair / Gyno
Cardio / Organ
Practical
Pharmacokinetics
Half-lifeFree: ~10 min | Varies by ester
RouteInjectable (esterified) / Transdermal / Implant
AromatizesYes — via CYP19A1 (aromatase)
HPTA SuppressionDose-dependent; significant at supraphysiological levels
Detection WindowTestosterone cypionate/enanthate: ~3 months | Propionate: ~2 weeks | Undecanoate: up to 6 months | Endogenous T/E ratio test can flag use
Anabolic / Androgenic Index
Anabolic
100
Androgenic
100
Testosterone = 100/100 reference. In vitro binding assay values; in vivo effects differ.
Mechanism of Action
Testosterone binds the androgen receptor (AR) with high affinity. The T–AR complex translocates to the nucleus acting as a transcription factor binding androgen response elements (AREs), upregulating protein synthesis genes (IGF-1, follistatin), nitrogen retention, and satellite cell activation. Converted peripherally to DHT via 5α-reductase amplifying androgenic effects, and to estradiol (E2) via aromatase producing both beneficial and adverse estrogenic effects.
Key Benefits
  • Dose-dependent skeletal muscle hypertrophy via AR-mediated protein synthesis
  • Increased IGF-1 and GH axis potentiation
  • Enhanced nitrogen retention and anti-catabolic signalling
  • Improved erythropoiesis (EPO upregulation → higher RBC/haematocrit)
  • Improved bone mineral density
  • Libido, mood, and cognitive function at physiological levels
Risks & Adverse Effects
  • HPTA suppression → testicular atrophy, LH/FSH suppression
  • Estrogen conversion → gynecomastia, water retention
  • DHT conversion → androgenic alopecia (genetically predisposed), BPH risk
  • LVH risk with chronic supraphysiological use
  • Erythrocytosis → elevated haematocrit → thrombosis risk
  • Dyslipidaemia: HDL suppression, LDL elevation
Monitoring Biomarkers
Total TFree TSHBGE2 (sensitive)LHFSHHaematocritRBCHDLLDLPSALFTs
💈 Hair Loss Risk
Hair Loss RiskMODERATE
Converted to DHT via 5α-reductase (SRD5A2) in scalp follicles. DHT binds follicular AR with 5× greater affinity than testosterone, shortening anagen phase and causing miniaturisation. Risk scales with dose.
✓ Mitigation Available: Finasteride (1mg/day) or dutasteride (0.5mg/day) block T→DHT conversion effectively. Topical minoxidil additive. Target DHT suppression with dutasteride for maximum protection.
⚠ Gynecomastia Risk
Gyno RiskMODERATE
Aromatizes via CYP19A1 to estradiol. E2 binds ERα in breast tissue → ductal proliferation. Risk scales non-linearly with dose.
Management: AI (anastrozole 0.5mg EOD, exemestane 12.5mg EOD) or SERMs (tamoxifen 20mg/day, raloxifene 60mg/day). Target E2 20–30 pg/mL. Over-suppression causes joint pain, libido loss, and worsens lipids.
♥ Cardiovascular Risk
Cardiovascular RiskMODERATE
LVH risk dose/duration dependent. HDL suppression proportional to dose. Haematocrit elevation → blood viscosity → thrombosis risk. Polycythaemia risk with long-term TRT.
Monitoring & Mitigation: Monitor BP, lipid panel, haematocrit every 3–6 months. Donate blood or therapeutic phlebotomy if haematocrit >52%. Aerobic exercise partially offsets LVH progression.
🫀 Organ Stress
Organ StressLOW
No hepatic burden (injectable form). Renal stress only with extreme doses/duration via haematocrit-driven hyperviscosity. Testicular atrophy from HPTA suppression — reversible in most cases.
Protocol: Safest on liver of all AAS when used as injectable. Prostate monitoring (PSA) essential with long-term use. HCG during long cycles preserves testicular volume and function.
PCT Required
Yes — HPTA suppression is significant. See PCT tab for protocol.
Female Risk
Female Virilisation RiskMODERATE
Virilisation possible at supraphysiological doses — voice deepening (irreversible), clitoral enlargement, acne, hair growth. Menstrual disruption. Low-dose TRT in women is clinically used and relatively safe with monitoring.
Counterfeit Risk
Counterfeit / Purity RiskLOW
Testosterone esters are the most commonly produced and generally reliable UGL compound. Concentration mislabelling (e.g. 200mg/mL labelled as 250mg/mL) is the most common issue rather than substitution.
Compound Relationship Map — Shared Mechanisms & Side Effect Clusters
Aromatize → Estrogen conversion
All Testosterone Esters Dianabol Nandrolone (low)
17α-Alkylated → Hepatotoxicity
Stanozolol Oxandrolone Dianabol Oxymetholone Halotestin
Progestogenic → Prolactin/Gyno
Trenbolone Nandrolone Decanoate NPP
DHT-Class → No 5α-RI Protection
Masteron Stanozolol Oxandrolone Proviron Halotestin
Severe Cardiovascular Risk
Trenbolone Stanozolol Halotestin Oxymetholone
Hair-Safest Injectables
Nandrolone Decanoate NPP Test Propionate + 5α-RI
PHARMACOLOGICAL REFERENCE ONLY. This knowledge graph is an educational tool. It does not constitute medical advice, dosing guidance, or endorsement of use. All compounds listed are controlled substances in numerous jurisdictions. Any therapeutic use requires medical supervision and prescription.
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